Using CHP to Understand Cardiac Fibrosis Pathology and Therapeutic Efficacy
Following a Myocardial Infarction (MI), the heart relies on myofibroblasts to deposit collagen, creating a vital "patch" to maintain structural integrity. However, when this process becomes chronic, excessive collagen deposition leads to cardiac fibrosis—a primary driver of heart failure. For researchers, the goal is to find therapies that limit this pathological remodeling without compromising...
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